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These types of reports have merit only if one already believes that TRT increases the risk of prostate cancer, since they show association but not causation, best sale steroids, steroids best sale. Since prostate cancer is so common, how does one know that TRT had anything at all to do with the subsequent identification of prostate cancer? It is an everyday occurrence for urologists to observe a sudden rise in PSA or a change on digital rectal exam that may trigger a biopsy. Since most of these changes occur without any known precipitating cause, any assertion that TRT caused the prostate change requires some evidence that TRT actually increased the rate of cancer in these men. Without this type of information, case reports or series provide no useful information at all. Indeed, it is even possible that the overall rate of cancer was decreased in these same urology practices among men receiving TRT. Clinical trials with TRT What do clinical trials actually tell us about the risk of prostate cancer among men receiving TRT? These effects include the growth and maturation of the prostate, seminal vesicles, penis, and scrotum the development of male hair distribution, such as facial, pubic, chest, and axillary hair laryngeal enlargement vocal cord thickening alterations in body musculature and fat distribution. Male hypogonadism results from insufficient secretion of testosterone and is characterized by low serum testosterone concentrations. Symptoms associated with male hypogonadism include decreased sexual desire with or without impotence, fatigue and loss of energy, mood depression, regression of secondary sexual characteristics, and osteoporosis. Hypogonadism is a risk factor for osteoporosis in men. Drugs in the androgen class also promote retention of nitrogen, sodium, potassium, phosphorus, and decreased urinary excretion of calcium. Androgens have been reported to increase protein anabolism and decrease protein catabolism.
Although one must be cautious in comparing one-year data to three–year data, there clearly was no dramatic rise in prostate cancers in this highrisk group receiving TRT. Longitudinal studies of endogenous T levels and prostate cancer risk Longitudinal studies with frozen sera represent one of the more powerful types of studies to investigate the potential etiologic effect of hormone levels on subsequent development of a disease such as prostate cancer, steroids 40 over anabolic. At least 12 such longitudinal population-based studies have been performed looking at the relationship of endogenous T levels (as well as other hormones) to prostate cancer, involving tens of thousands of men. At the end of the study, a cohort had developed prostate cancer, and the remainder did not. Blood samples from these men from years earlier were then compared to see whether testosterone levels differed between these two groups, steroids anabolic 40 over. Not one of the twelve studies demonstrated a difference in T levels between men who did and did not develop cancer.
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